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It suppresses pathological axonal sprouting in dorsal root ganglia (DRG) by downregulating estradiol (E 2 ), angiotensin II (Ang II), and angiotensin II type 2 receptor (AT 2 R) expression, which are key mediators of neurogenic inflammation and peripheral sensitization, resulting in a dose-dependent increase in mechanical withdrawal threshold and clear improvement of chronic pelvic hyperalgesia (Yin et al., 2024)

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The combination is synergistic because BPC-157 and TB-500 address complementary stages of the healing cascade: Phase 1, Inflammation (Days 1-5): TB-500 suppresses the inflammatory cytokine response while BPC-157 begins upregulating growth factor signaling at the injury site