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In addition, the Connectivity Map gene profiling software developed by the Broad Institute, showed that out of several thousand biological molecules, the naturally occurring peptide glycyl-L-histidyl-L-lysine (GHK), in its Cu-bound form prevented impaired TGFβ1 signaling [4], a pathway associated with Aβ deposition and neurofibrillary tangle formation [5]

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doi: 10.1080/10408449991349186