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Following chronic liver injury, HSCs proliferate, lose their vitamin A and undergo a major phenotypical transformation to α-smooth muscle actin (α-SMA) positive activated HSCs, which produce a wide variety of collagenous and non-collagenous extracellular matrix (ECM) proteins [168]

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Finally, group 10 acted as a vehicle control for Dihexa in group 8 and received local MSC and DMSO via hydrogel, and intravenous injection of MSC, and an injection into the gastrocnemius muscle with MSC and DMSO [2]